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357,361,371,372,373 Consistently, amygdalar axonal projections and neuronal activation are defective in Tbr1(+/) mice, but these defects are ameliorated by infusion of an NMDA receptor agonist (D-cycloserine)

no changes in PDH phosphorylation were observed (Fig

However, the formation of selenoxide could be observed when the cyclization is blocked by replacing the N-H moiety in compound with an N-Et group

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