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Indeed, tyrosine-972 in the NPEY motif of IR (numbering relative to human IR-B or +exon 11 isoform), which is phosphorylated following ligand stimulation, serves as a docking site for proteins with a PTB domain, such as IRS-1 and Shc, and mutation of this residue markedly decreases phosphorylation of these receptor substrates and activation of downstream signaling (16, 17)
Insulin may be less effective, or the liver may release too much glucose, causing blood sugar to remain elevated
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