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Ultrafine silicon dioxide nanoparticles cause lung epithelial cells apoptosis via oxidative stress-activated PI3K/Akt-mediated mitochondria- and endoplasmic reticulum stress-dependent signaling pathways

Their intrinsic biodegradability, biocompatibility, tunable particle size, facile surface functionalization, and capacity to protect labile cargos from acidic and enzymatic degradation enable enhanced stability, targeted release, and improved bioavailability of polyphenols, peptides, and probiotics

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Stress is easily induced with some common causes being: changes in diet, heavy work or training, temperature extremes, breeding, transportation, worming, vaccinations and changes to environment