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IGF-1 LR3 retains the ability to bind IGF-1 and insulin receptors but has reduced binding affinity to insulin-like growth factor-binding proteins (IGFBPs)

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These synergy studies established the mechanistic principle underpinning the GHRH analogue + GHS-R1a agonist combination format demonstrating that dual-pathway somatotroph co-stimulation is not merely additive but produces signal amplification through convergent intracellular mechanisms