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These findings illustrate the therapeutic potential of dual-pathway intervention, although the complex interplay between autophagy, ferroptosis and various immune cell populations requires further mechanistic elucidation to optimize combination strategies

This mechanistic insight has prompted the exploration of ferroptosis as a therapeutic target, given its potential contribution to dopaminergic neuronal loss and disease progression

Impaired brain insulin signaling has emerged as a feature that contributes to neuronal dysfunction in both AD and PD, leading to strategies aiming at restoring this pathway in the brain

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