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Further studies are required to clarify the complex interactions between GLP-1 RAs and various neuropsychiatric processes

Glucose is a substrate for lipogenesis, and either dietary intake of glucose and fructose or hyperglycemia due to T2DM-associated insulin resistance can lead to hepatic de novo lipogenesis

Alongside liraglutide, semaglutide has been shown to effectively improve motor impairments, rescue tyrosine hydroxylase (TH) levels, reduce -synuclein accumulation, chronic inflammation response in the brain and lipid peroxidation, inhibit mitochondrial mitophagy signaling and protect dopaminergic neurons in the substantia nigra and striatum in MPTP mouse models of PD [81]

R., & Apovian, C