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Furthermore, AS-IV may activate the PI3K/Akt signaling pathway to suppress oxidative stress, contributing to the amelioration of DOX-induced cardiac dysfunction (Luo et al., 2021b)

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When B cells were stimulated with a different Fc-binding protein, Staphylococcus aureus protein A (SpA), CD69 and CD86 were upregulated to a similar degree as compared to -BCR (Fig EV5A)