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The divergence in the effect of mutagenesis of residues comprising the TM binding pocket, even where interactions in the static consensus structures were similar, confirmed that static visualisation of complexes is insufficient to fully understand binding and activation mechanisms, and suggests that the dynamics of peptide-receptor engagement likely play a critical role

In plain terms: GHK-Cu helps ensure the repaired tissue is stronger than it was before

In autoimmune disease, the balance tilts heavily toward M1