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Many other cofactors can act synergically with hyperammonemia, to produce or aggravate HE, but experts do not consider them to be exclusive causes of HE, on their own or in isolation, i.e., in the absence of hyperammonemia.10 Patients with portosystemic shunts (PSSs) and no liver disease can also present with hyperammonemia-associated brain dysfunction despite having a functional or normal liver11

CYP24A1 is overexpressed in keloid keratinocytes and its inhibition alters profibrotic gene expression

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La formulation de ces antioxydants sous forme de gouttes oculaires prsente une mthode dadministration localise et cible, garantissant que des concentrations efficaces atteignent la rtine