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In the LPS-induced acutelung injury mouse model, treatment of GHK-Cu also significantly suppressed TNF- and IL-6 expression with a significant decrease of both NF-B p65 and p38 MAPK signaling, suggesting that GHK-Cu is useful for the development of novel anti-inflammatory therapies [22]

Animal model research demonstrates that BPC-157 administration reduces tissue inflammation scores by 40-60% across diverse experimental systems including inflammatory bowel disease, musculoskeletal injury, and neuroinflammation models