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However, in 2001, a handful of groundbreaking research discoveries confirmed that the Ca 2+ ionophore ionomycin could trigger an apoptosis-like cell death in erythrocytes associated with the appearance of apoptotic signs, including cell shrinkage, membrane vesiculation, or PS exposure [22, 23]

(4) Standard therapy combines atropine, which antagonizes muscarinic receptor overstimulation, with pralidoxime (2-PAM), which reactivates uninhibited or non-aged AChE