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By activating FOXO, SIRT1 increases the expression of antioxidant enzymes like MnSOD and CAT, which counteract the formation of ROS [126,127]

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Its mechanism is based on increasing the expression of neurotrophic factors such as BDNF (Brain-Derived Neurotrophic Factor), which translates into: DIHEXA was developed by scientists at Washington State University as a potential therapeutic agent, originally studied for the treatment of neurodegenerative diseases such as Alzheimer's and Parkinson's