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Telomerase is normally active only in germline cells, stem cells, and cancer cells

Font-Burgada, D

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Overall, these data suggest that NR4a1 overexpression alone does not exacerbate TAC-induced pathological cardiac remodeling but counteracts the therapeutic effect of Sema, further confirming that NR4a1 is a target of Sema in pressure overload-induced cardiac remodeling and dysfunction