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Dayer, J

When pentadecapeptide BPC 157 therapy was given in already advanced stage of paracetamol toxicity, such as at 3 hours after initial paracetamol application, only mild liver lesions ( Table 3, Fig

Future work will use longitudinal profiling and single-cell sequencing to assess cell-type-specific GSH roles 5 Conclusion In conclusion, by leveraging multi-omics integration, the present study identified GSH metabolism as a pivotal node in SAE, while providing a comprehensive molecular framework for SAE pathogenesis, and linking microglia-driven neuroinflammation to hippocampal oxidative damage