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10.1016/j.pneurobio.2020.101969 29 KlausenM

Extended follow-up studies are therefore essential to determine whether the histologic improvements translate into durable reductions in cirrhosis, HCC, and all-cause mortality

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IGF-DES and IGFBP Biology Research Research using radiolabelled des(1-3)IGF-1 as a probe confirmed that IGFBP-3 is as potent as unlabelled IGF-1 in displacing it from cell surface binding providing evidence that IGFBP-3 can modulate IGF-1R access via a cellular mechanism distinct from simple extracellular sequestration, a key finding in IGFBP-IGF-1R interaction research