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In type 2 diabetes-related cervical cancer tissues, a high-glucose microenvironment induces the upregulation of PSMA2, phosphorylated IB, and phosphorylated P65 expression, thereby promoting tumor progression, while Ex-4 can effectively inhibit this pathological process [120]

Furthermore, the prevalence of insulin resistance, a major causative factor in the early development of type 2 diabetes and an independent risk factor for cardiovascular disease and the metabolic syndrome X, is even more widespread [3][4][5]