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The mechanistic core of the finding was that Dihexa activated the PI3K/AKT signaling pathway in the brains of the APP/PS1 mice, and that the PI3K inhibitor wortmannin reversed Dihexas anti-inflammatory and anti-apoptotic effects [5]

For brain-targeting compounds, where the goal is modulating delicate neurotransmitter systems and growth factor pathways, impurities are not just unwanted, they are potentially dangerous

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